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INDEXED BY
 
CONFERENCE TO BE HELD IN
Meiji University, Tokyo, Japan
SPONSORED BY
SUPPORTED BY
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KEYNOTE
SPEAKER
Keynote Speaker I
Prof. Walter Herzog
University of Calgary, Canada
Dr. Herzog did his undergraduate training
in Physical Education at the Federal Technical Institute in
Zurich, Switzerland (1979), completed his doctoral research
in Biomechanics at the University of Iowa (USA) in 1985, and
completed postdoctoral fellowships in Neuroscience and
Biomechanics in Calgary, Canada in 1987. Currently, Dr.
Herzog is a Professor of Biomechanics with appointments in
Kinesiology, Medicine, Engineering, and Veterinary Medicine,
holds the Canada Research Chair for Cellular and Molecular
Biomechanics, and is appointed the Killam Memorial Chair for
Inter-Disciplinary Research at the University of Calgary.
His research interests are in musculoskeletal biomechanics
with emphasis on mechanisms of muscle contraction focusing
on the role of the structural protein titin, and the
biomechanics of joints focusing on mechanisms of onset and
progression of osteoarthritis. Dr. Herzog is the recipient
of the Borelli Award from the American Society of
Biomechanics, the Career Award from the Canadian Society for
Biomechanics, the Dyson Award from the International Society
of Biomechanics in Sports, the Muybridge Award from the
International Society of Biomechanics, and recently received
the Killam Prize in Engineering from the Canada Council for
the Arts for his contributions to Biomedical research. He is
the past president of the International, American and
Canadian Societies for Biomechanics. He was inducted into
the Royal Society of Canada in 2013.
Speech Title: "The Forgotten Filament in
Muscle Contraction"
Abstract: The sliding filament and
cross-bridge theories of muscle contraction have dominated
the thinking in muscle contraction research for more than
half a century. These theories rely on force production by
the contractile filaments actin and myosin exclusively.
However, many experimental observations on skeletal muscle
properties cannot be explained using these theories. For
example, the cross-bridge theory cannot explain the residual
force enhancement property of skeletal muscle, nor can it
account for the stability of myosin in the centre of
sarcomeres. In 2002, we discovered that passive forces in an
active muscle are much greater than the passive forces in a
passive muscle. We called this observation “passive force
enhancement”. In 2008, we demonstrated that this increased
passive force is uniquely associated with the filamentous,
sarcomeric protein titin. We then showed that titin is a
spring protein whose stiffness increases with muscle
activation, thus providing more force when a muscle is
actively stretched compared to when it is passively
stretched. The molecular mechanisms of this increase in
titin stiffness with activation remain unclear, but I will
present the latest findings from our group on how titin
might regulate active force in skeletal muscle contraction.
Keynote Speaker II
Prof. Jyh-Ping Chen
Chang Gung University, Taiwan
Dr. Jyh-Ping Chen has been a professor in
Chemical and Materials Engineering at Chang Gung University
since 1997. He is currently a research fellow in the
Department of Plastic and Reconstructive Surgery, Chang Gung
Memorial Hospital and holds joint appointments as professor
in Ming Chi University of Technology and Chang Gung
University of Science and Technology, Taiwan. He received
his PhD in Chemical Engineering from Pennsylvania State
University. He established the Graduate Institute of
Biochemical and Biomedical Engineering in 2002 and served as
the first director of the institute till 2006. He was the
chairman of the Chemical and Materials Engineering
Department from 2006 to 2014. Professor Chen has published
over 150 papers in SCI journals with more than 5500
citations. He is a guest editor or editorial board member
for 15 international journals and a peer reviewer for more
than 50 reputed journals. His current research interests
include biomaterials, tissue engineering and drug delivery.
INVITED
SPEAKER
Invited Speaker I
Assoc. Prof. Jennifer Chua Dela Cruz
Mapua University, Philippine
Earned my PhD ECE (Electronics and
Communications Engineering) and MSECE from De LaSalle
University, and BSECE from Mapua University. Currently the
Research Coordinator of the School of EECE and faculty
member both of undergraduate and graduate studies of Mapua
University. Have presented and published more than 60
research papers in the field of Electronics, Computing and
Engineering. Has on-going research collaborations with
Department of Science and Technology and ASEAN IVO.
Conducting consultancies as Professional Electronics
Engineer and also as an ASEAN Engineer. Serving as program
evaluator and accreditor of Electronics Engineering program
of local Universities and Colleges for Commission on Higher
Education and Philippine Technological Council. Active
volunteer and Senior Member of IEEE, 2017 and 2018 Chairman
of IEEE (Institute of Electrical and Electronics Engineers)
Philippines Section and founder and acting Chairman IEEE PS
Women in Engineering and IEEE PS Education Society Chapter.
Speech Title: "Portable Nuclear and Cortical Eye Cataract Detection using Image Processing"
Abstract: Early detection and treatment
of cataract can prevent worse effects in eyesight, such as
blurred vision and blindness. However, portability is an
issue with the machines that are used for detecting eye
cataracts. The focus of this paper will be the detection and
classification of cataract using image processing algorithms
which are Local Ridge Enhancement, Sobel Algorithm,
Directional Coding, and Back Propagation Neural Network. The
mobile application that shall serve as this study’s output
will display whether the user has an eye cataract or not. If
an eye cataract is detected, the app shall only display the
type of cataract and the severity grade of the eye cataract.
Based on the data gathered and the doctor’s assessment, this
paper resulted in a 100% accuracy on detecting and
classifying Normal Eyes, 80.00% on Nuclear Mild Cataract,
85.00% on Nuclear Severe Cataract, 80.00% on Cortical Mild
Cataract, and 93.33% on Cortical Severe Cataract.
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